2016

  1. Martin MD, and VP Badovinac. Sifting through CD8 T cell memory. Immunity 45: 1184-1186 (2016) [PubMed
  2. He B, S Xing, C Chen, P Gao, L Teng, Q Shan, JA Gullicksrud, MD Martin, S Yu, JT Harty, VP Badovinac, K Tan, and HH Xue.  CD8 T cells utilize highly dynamic enhancer repertoires and regulatory circuitry in response to infections. Immunity 45: 1341-1354 (2016) [PubMed]
  3. Strother RK, DB Danahy, DI Kotov, TA Kucaba, ZR Zacharias, TS Griffith, KL Legge, and VP Badovinac.  Polymicrobial sepsis diminishes dendritic cell numbers and function directly contributing to impaired primary CD8 T cell responses in vivo. J Immunol 197: 4301-4311 (2016) [PubMed]
  4. Janowski AM, OR Colegio, EE Hornick, JM McNiff, MD Martin, VP Badovinac, LA Norian, W Zhang, SL Cassel, and FS Sutterwala. NLRC4 suppresses melanoma tumor progression independently of Inflammasome activation. J Clin Invest 126: 3917-3928 (2016) [PubMed]
  5. Danahy DB, RK Strother, VP Badovinac, and TS Griffith. Clinical and experimental sepsis impair T cell-mediated immunity. Crit Rev Immunol 36: 57-74 (2016) [PubMed]
  6. Cabrera-Perez J, JC Babcock, T Dileepan, KA Murphy, TA Kucaba, VP Badovinac, and TS Griffith. Gut microbial membership modulates CD4 T cell reconstitution and function after sepsis. J  Immunol  197: 1692-1698 (2016) [PubMed]

2015

  1. Martin MD, and VP Badovinac. Antigen-dependent and –independent contributions to primary memory CD8 T cell activation and protection following infection. Sci Rep 5: 18022 (2015) [PubMed]
  2. Martin MD, MT Kim, Q Shan, R Sompallaie, HH Xue, JT Harty, and VP Badovinac. Phenotypic and functional alterations in circulating memory CD8 T cells with time after primary infection. PLoS Pathog 11: e1005219 (2015) [PubMed]
  3. Khan SH, MD Martin, GR Starbeck-Miller, HH Xue, JT Harty, and VP Badovinac. The timing of stimulation and IL-2 signaling regulate secondary CD8 T cell responsesPLoS Pathog 11: e1005199 (2015) [PubMed]
  4. Kim MT, MJ Richer, BP Gross, LA Norian, VP Badovinac, and JT Harty.  Enhancing dendritic cell-based immunotherapy with IL-2/mAb complexes for control of established tumors.  J Immunol 195: 4537-4544 (2015) [PubMed]
  5. Condotta SA, SH Khan, D Rai, TS Griffith, and VP Badovinac. Poly-microbial sepsis increases susceptibility to chronic viral infection and exacerbates CD8 T cell exhaustion. J Immunol 195: 116-125 (2015) [PubMed]
  6. Khan SH, and VP Badovinac. Listeria monocytogenes: a model pathogen to study antigen-specific memory CD8 T cell responses. Semin Immunopathol 37: 301-310 (2015) [PubMed]
  7. Cabrera-Perez J, SA Condotta, BR James, SW Kashem, EL Brincks, D Rai, TA Kucaba, VP Badovinac, and TS Griffith.  Alterations in Ag-specific naïve CD4 T cell precursors after sepsis impairs their responsiveness to pathogen challenge. J Immunol 194: 1609-1620 (2015) [PubMed]

2014

  1. Khan SH, EA Hemann, KL Legge, LA Norian, and VP Badovinac. Diet-induced obesity does not impact the maintenance of memory CD8 T cells. J Immunol 193: 5873-5882 (2014) [PubMed]
  2. Rai D, MD Martin, and VP Badovinac. The longevity of memory CD8 T cell responses after repetitive antigen-stimulations. J Immunol 192: 5652-5659 (2014) [PubMed]
  3. Duong S, SA Condotta, D Rai, MD Martin, TS Griffith, and VP Badovinac.  Poly-microbial sepsis alters Ag-dependent and –independent memory CD8 T cell functions. J Immunol 192, 3618-3625 (2014) [PubMed]
  4. ​Martin MD, and VP Badovinac. Influence of time and number of antigen encounters on memory CD8 T cell development. Immunol Res 59: 35-44 (2014) [PubMed]
  5. Starbeck-Miller GR, VP Badovinac, and JT Harty.  Cutting Edge: Expression of FcgRIIB tempers memory CD8 T cell function in vivo. J Immunol 192: 35-39 (2014) [PubMed]
  6. Cabrera-Perez J, SA Condotta, VP Badovinac, and TS Griffith. The impact of sepsis on CD4 T cell immunity. J Leuk Biol 96: 767-777 (2014) [PubMed]
  7. Markwart R, SA Condotta, RP Requardt, F Borken, K Schubert, C Weigel, M Bauer, TS Griffith, M Forster, FM Brunkhorst, VP Badovinac, and I Rubio. Immunosuppression after sepsis: systemic inflammation and sepsis induce a loss of naïve T-cells but no enduring cell-autonomous defects in T-cell functionPLoS One 9: e115094 (2014) [PubMed]

Complete Publication List at PubMed